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These impairments likely were related to thickening of the LV muscle wall in the AAS users, but these changes are variable among subjects. One study showed that AAS users have an increased amount of calcium deposited in LV muscle. This is associated with a greater number of myocardial infarctions but not with the more pronounced effect with the oral AAS (21). It is noteworthy that the effects of the AAS were seen within the first 2 weeks of initiating drug use. It is likely that these effects are caused by changes in the body's regulatory mechanism to prevent the AAS from inducing cardiac damage. The long-term effects are difficult to determine because of the many variables that influence AAS intake. For example, the subjects in the above study were young adults who began the study with moderate AAS intake. As the study progressed, it is likely that AAS intake became more severe. These long-term effects may explain why AAS users are more likely to develop cardiovascular disease. Cardiovascular disease is not the only effect of the AAS, but it is related in some respects. If you know about this link between the AAS and the AVD, make sure to read about this link. It also may explain why AAS users may be at an increased risk for cardiovascular disease. Related Article:
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